Expression of FTHL17 in cancer - Summary - The Human Protein Atlas

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FTHL17

TISSUE

CELL

PATHOLOGY

GENERAL INFORMATION
Gene name	FTHL17
Gene description	Ferritin, heavy polypeptide-like 17
Protein class	Predicted intracellular proteins
Predicted localization	Intracellular
Number of transcripts	1
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HUMAN PROTEIN ATLAS INFORMATION

RNA category

TCGA (cancer tissue):	Tissue enhanced (testis cancer)
HPA (cell line):	Not detected
HPA (normal tissue):	Tissue enriched (testis)

Protein evidence

Evidence at transcript level

ANTIBODY IHC RELIABILITY
Reliability score	Pending cancer tissue analysis.

PROGNOSTIC SUMMARY
Gene product is not prognostic.
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RNA EXPRESSION OVERVIEW
TCGA dataset RNA cancer category: Tissue enhanced (testis cancer)

PROTEIN EXPRESSION
Pending cancer tissue analysis
STAINING SUMMARY

GENE INFORMATION
Gene name	FTHL17 (HGNC Symbol)
Synonyms	CT38
Description	Ferritin, heavy polypeptide-like 17 (HGNC Symbol)
Entrez gene summary	This gene is orthologous to a mouse gene that encodes a ferritin heavy polypeptide-like protein and shows testis-specific expression. [provided by RefSeq, Oct 2009]
Chromosome	X
Cytoband	p21.2
Chromosome location (bp)	31071241 - 31072053
Protein evidence	Evidence at transcript level (all genes)
Ensembl	ENSG00000132446 (version 83.38)
Entrez gene	53940
UniProt	Q9BXU8 (UniProt - Evidence at transcript level)
neXtProt	NX_Q9BXU8
Antibodypedia	FTHL17 antibodies

PROTEIN BROWSER

FTHL17-001

PROTEIN INFORMATION
Splice variant	UniProt	Protein class	Gene ontology	Length & mass	Signal peptide (predicted)	Transmembrane regions (predicted)
FTHL17-001 ENSP00000368207 ENST00000359202	Q9BXU8 [Direct mapping] Ferritin heavy polypeptide-like 17 Show all	TMHMM predicted membrane proteins Predicted intracellular proteins Show all	GO:0005623 [cell] GO:0006826 [iron ion transport] GO:0006879 [cellular iron ion homeostasis] GO:0008199 [ferric iron binding] Show all	183 aa 21.1 kDa	No	0

Contact

contact@proteinatlas.org

The Project

The Human Protein Atlas

The Human Protein Atlas project is funded
by the Knut & Alice Wallenberg foundation.